Serveur d'exploration sur le peuplier

Attention, ce site est en cours de développement !
Attention, site généré par des moyens informatiques à partir de corpus bruts.
Les informations ne sont donc pas validées.

Basic self-disturbance predicts psychosis onset in the ultra high risk for psychosis "prodromal" population.

Identifieur interne : 002B89 ( Main/Exploration ); précédent : 002B88; suivant : 002B90

Basic self-disturbance predicts psychosis onset in the ultra high risk for psychosis "prodromal" population.

Auteurs : Barnaby Nelson [Australie] ; Andrew Thompson ; Alison R. Yung

Source :

RBID : pubmed:22349924

Descripteurs français

English descriptors

Abstract

INTRODUCTION

Phenomenological research indicates that disturbance of the basic sense of self may be a core phenotypic marker of schizophrenia spectrum disorders. Basic self-disturbance refers to a disruption of the sense of ownership of experience and agency of action and is associated with a variety of anomalous subjective experiences. In this study, we investigated the presence of basic self-disturbance in an "ultra high risk" (UHR) for psychosis sample compared with a healthy control sample and whether it predicted transition to psychotic disorder.

METHODS

Forty-nine UHR patients and 52 matched healthy control participants were recruited to the study. Participants were assessed for basic self-disturbance using the Examination of Anomalous Self-Experience (EASE) instrument. UHR participants were followed for a mean of 569 days.

RESULTS

Levels of self-disturbance were significantly higher in the UHR sample compared with the healthy control sample (P < .001). Cox regression indicated that total EASE score significantly predicted time to transition (P < .05) when other significant predictors were controlled for. Exploratory analyses indicated that basic self-disturbance scores were higher in schizophrenia spectrum cases, irrespective of transition to psychosis, than nonschizophrenia spectrum cases.

DISCUSSION

The results indicate that identifying basic self-disturbance in the UHR population may provide a means of further "closing in" on individuals truly at high risk of psychotic disorder, particularly of schizophrenia spectrum disorders. This may be of practical value by reducing inclusion of "false positive" cases in UHR samples and of theoretical value by shedding light on core phenotypic features of schizophrenia spectrum pathology.


DOI: 10.1093/schbul/sbs007
PubMed: 22349924
PubMed Central: PMC3494062


Affiliations:


Links toward previous steps (curation, corpus...)


Le document en format XML

<record>
<TEI>
<teiHeader>
<fileDesc>
<titleStmt>
<title xml:lang="en">Basic self-disturbance predicts psychosis onset in the ultra high risk for psychosis "prodromal" population.</title>
<author>
<name sortKey="Nelson, Barnaby" sort="Nelson, Barnaby" uniqKey="Nelson B" first="Barnaby" last="Nelson">Barnaby Nelson</name>
<affiliation wicri:level="4">
<nlm:affiliation>Orygen Youth Health Research Centre, University of Melbourne, 35 Poplar Road (Locked Bag 10), Parkville, Victoria 3052, Australia. nelsonb@unimelb.edu.au</nlm:affiliation>
<country xml:lang="fr">Australie</country>
<wicri:regionArea>Orygen Youth Health Research Centre, University of Melbourne, 35 Poplar Road (Locked Bag 10), Parkville, Victoria 3052</wicri:regionArea>
<orgName type="university">Université de Melbourne</orgName>
<placeName>
<settlement type="city">Melbourne</settlement>
<region type="état">Victoria (État)</region>
</placeName>
</affiliation>
</author>
<author>
<name sortKey="Thompson, Andrew" sort="Thompson, Andrew" uniqKey="Thompson A" first="Andrew" last="Thompson">Andrew Thompson</name>
</author>
<author>
<name sortKey="Yung, Alison R" sort="Yung, Alison R" uniqKey="Yung A" first="Alison R" last="Yung">Alison R. Yung</name>
</author>
</titleStmt>
<publicationStmt>
<idno type="wicri:source">PubMed</idno>
<date when="2012">2012</date>
<idno type="RBID">pubmed:22349924</idno>
<idno type="pmid">22349924</idno>
<idno type="doi">10.1093/schbul/sbs007</idno>
<idno type="pmc">PMC3494062</idno>
<idno type="wicri:Area/Main/Corpus">002B32</idno>
<idno type="wicri:explorRef" wicri:stream="Main" wicri:step="Corpus" wicri:corpus="PubMed">002B32</idno>
<idno type="wicri:Area/Main/Curation">002B32</idno>
<idno type="wicri:explorRef" wicri:stream="Main" wicri:step="Curation">002B32</idno>
<idno type="wicri:Area/Main/Exploration">002B32</idno>
</publicationStmt>
<sourceDesc>
<biblStruct>
<analytic>
<title xml:lang="en">Basic self-disturbance predicts psychosis onset in the ultra high risk for psychosis "prodromal" population.</title>
<author>
<name sortKey="Nelson, Barnaby" sort="Nelson, Barnaby" uniqKey="Nelson B" first="Barnaby" last="Nelson">Barnaby Nelson</name>
<affiliation wicri:level="4">
<nlm:affiliation>Orygen Youth Health Research Centre, University of Melbourne, 35 Poplar Road (Locked Bag 10), Parkville, Victoria 3052, Australia. nelsonb@unimelb.edu.au</nlm:affiliation>
<country xml:lang="fr">Australie</country>
<wicri:regionArea>Orygen Youth Health Research Centre, University of Melbourne, 35 Poplar Road (Locked Bag 10), Parkville, Victoria 3052</wicri:regionArea>
<orgName type="university">Université de Melbourne</orgName>
<placeName>
<settlement type="city">Melbourne</settlement>
<region type="état">Victoria (État)</region>
</placeName>
</affiliation>
</author>
<author>
<name sortKey="Thompson, Andrew" sort="Thompson, Andrew" uniqKey="Thompson A" first="Andrew" last="Thompson">Andrew Thompson</name>
</author>
<author>
<name sortKey="Yung, Alison R" sort="Yung, Alison R" uniqKey="Yung A" first="Alison R" last="Yung">Alison R. Yung</name>
</author>
</analytic>
<series>
<title level="j">Schizophrenia bulletin</title>
<idno type="eISSN">1745-1701</idno>
<imprint>
<date when="2012" type="published">2012</date>
</imprint>
</series>
</biblStruct>
</sourceDesc>
</fileDesc>
<profileDesc>
<textClass>
<keywords scheme="KwdEn" xml:lang="en">
<term>Adolescent (MeSH)</term>
<term>Case-Control Studies (MeSH)</term>
<term>Disease Progression (MeSH)</term>
<term>Female (MeSH)</term>
<term>Humans (MeSH)</term>
<term>Longitudinal Studies (MeSH)</term>
<term>Male (MeSH)</term>
<term>Prodromal Symptoms (MeSH)</term>
<term>Prognosis (MeSH)</term>
<term>Proportional Hazards Models (MeSH)</term>
<term>Psychotic Disorders (diagnosis)</term>
<term>Psychotic Disorders (psychology)</term>
<term>Regression Analysis (MeSH)</term>
<term>Schizophrenia (diagnosis)</term>
<term>Schizophrenic Psychology (MeSH)</term>
<term>Self Concept (MeSH)</term>
<term>Young Adult (MeSH)</term>
</keywords>
<keywords scheme="KwdFr" xml:lang="fr">
<term>Adolescent (MeSH)</term>
<term>Analyse de régression (MeSH)</term>
<term>Concept du soi (MeSH)</term>
<term>Femelle (MeSH)</term>
<term>Humains (MeSH)</term>
<term>Jeune adulte (MeSH)</term>
<term>Modèles des risques proportionnels (MeSH)</term>
<term>Mâle (MeSH)</term>
<term>Pronostic (MeSH)</term>
<term>Psychologie des schizophrènes (MeSH)</term>
<term>Schizophrénie (diagnostic)</term>
<term>Symptômes prodromiques (MeSH)</term>
<term>Troubles psychotiques (diagnostic)</term>
<term>Troubles psychotiques (psychologie)</term>
<term>Études cas-témoins (MeSH)</term>
<term>Études longitudinales (MeSH)</term>
<term>Évolution de la maladie (MeSH)</term>
</keywords>
<keywords scheme="MESH" qualifier="diagnosis" xml:lang="en">
<term>Psychotic Disorders</term>
<term>Schizophrenia</term>
</keywords>
<keywords scheme="MESH" qualifier="diagnostic" xml:lang="fr">
<term>Schizophrénie</term>
<term>Troubles psychotiques</term>
</keywords>
<keywords scheme="MESH" qualifier="psychologie" xml:lang="fr">
<term>Troubles psychotiques</term>
</keywords>
<keywords scheme="MESH" qualifier="psychology" xml:lang="en">
<term>Psychotic Disorders</term>
</keywords>
<keywords scheme="MESH" xml:lang="en">
<term>Adolescent</term>
<term>Case-Control Studies</term>
<term>Disease Progression</term>
<term>Female</term>
<term>Humans</term>
<term>Longitudinal Studies</term>
<term>Male</term>
<term>Prodromal Symptoms</term>
<term>Prognosis</term>
<term>Proportional Hazards Models</term>
<term>Regression Analysis</term>
<term>Schizophrenic Psychology</term>
<term>Self Concept</term>
<term>Young Adult</term>
</keywords>
<keywords scheme="MESH" xml:lang="fr">
<term>Adolescent</term>
<term>Analyse de régression</term>
<term>Concept du soi</term>
<term>Femelle</term>
<term>Humains</term>
<term>Jeune adulte</term>
<term>Modèles des risques proportionnels</term>
<term>Mâle</term>
<term>Pronostic</term>
<term>Psychologie des schizophrènes</term>
<term>Symptômes prodromiques</term>
<term>Études cas-témoins</term>
<term>Études longitudinales</term>
<term>Évolution de la maladie</term>
</keywords>
</textClass>
</profileDesc>
</teiHeader>
<front>
<div type="abstract" xml:lang="en">
<p>
<b>INTRODUCTION</b>
</p>
<p>Phenomenological research indicates that disturbance of the basic sense of self may be a core phenotypic marker of schizophrenia spectrum disorders. Basic self-disturbance refers to a disruption of the sense of ownership of experience and agency of action and is associated with a variety of anomalous subjective experiences. In this study, we investigated the presence of basic self-disturbance in an "ultra high risk" (UHR) for psychosis sample compared with a healthy control sample and whether it predicted transition to psychotic disorder.</p>
</div>
<div type="abstract" xml:lang="en">
<p>
<b>METHODS</b>
</p>
<p>Forty-nine UHR patients and 52 matched healthy control participants were recruited to the study. Participants were assessed for basic self-disturbance using the Examination of Anomalous Self-Experience (EASE) instrument. UHR participants were followed for a mean of 569 days.</p>
</div>
<div type="abstract" xml:lang="en">
<p>
<b>RESULTS</b>
</p>
<p>Levels of self-disturbance were significantly higher in the UHR sample compared with the healthy control sample (P < .001). Cox regression indicated that total EASE score significantly predicted time to transition (P < .05) when other significant predictors were controlled for. Exploratory analyses indicated that basic self-disturbance scores were higher in schizophrenia spectrum cases, irrespective of transition to psychosis, than nonschizophrenia spectrum cases.</p>
</div>
<div type="abstract" xml:lang="en">
<p>
<b>DISCUSSION</b>
</p>
<p>The results indicate that identifying basic self-disturbance in the UHR population may provide a means of further "closing in" on individuals truly at high risk of psychotic disorder, particularly of schizophrenia spectrum disorders. This may be of practical value by reducing inclusion of "false positive" cases in UHR samples and of theoretical value by shedding light on core phenotypic features of schizophrenia spectrum pathology.</p>
</div>
</front>
</TEI>
<pubmed>
<MedlineCitation Status="MEDLINE" Owner="NLM">
<PMID Version="1">22349924</PMID>
<DateCompleted>
<Year>2013</Year>
<Month>05</Month>
<Day>16</Day>
</DateCompleted>
<DateRevised>
<Year>2018</Year>
<Month>11</Month>
<Day>13</Day>
</DateRevised>
<Article PubModel="Print-Electronic">
<Journal>
<ISSN IssnType="Electronic">1745-1701</ISSN>
<JournalIssue CitedMedium="Internet">
<Volume>38</Volume>
<Issue>6</Issue>
<PubDate>
<Year>2012</Year>
<Month>Nov</Month>
</PubDate>
</JournalIssue>
<Title>Schizophrenia bulletin</Title>
<ISOAbbreviation>Schizophr Bull</ISOAbbreviation>
</Journal>
<ArticleTitle>Basic self-disturbance predicts psychosis onset in the ultra high risk for psychosis "prodromal" population.</ArticleTitle>
<Pagination>
<MedlinePgn>1277-87</MedlinePgn>
</Pagination>
<ELocationID EIdType="doi" ValidYN="Y">10.1093/schbul/sbs007</ELocationID>
<Abstract>
<AbstractText Label="INTRODUCTION" NlmCategory="BACKGROUND">Phenomenological research indicates that disturbance of the basic sense of self may be a core phenotypic marker of schizophrenia spectrum disorders. Basic self-disturbance refers to a disruption of the sense of ownership of experience and agency of action and is associated with a variety of anomalous subjective experiences. In this study, we investigated the presence of basic self-disturbance in an "ultra high risk" (UHR) for psychosis sample compared with a healthy control sample and whether it predicted transition to psychotic disorder.</AbstractText>
<AbstractText Label="METHODS" NlmCategory="METHODS">Forty-nine UHR patients and 52 matched healthy control participants were recruited to the study. Participants were assessed for basic self-disturbance using the Examination of Anomalous Self-Experience (EASE) instrument. UHR participants were followed for a mean of 569 days.</AbstractText>
<AbstractText Label="RESULTS" NlmCategory="RESULTS">Levels of self-disturbance were significantly higher in the UHR sample compared with the healthy control sample (P < .001). Cox regression indicated that total EASE score significantly predicted time to transition (P < .05) when other significant predictors were controlled for. Exploratory analyses indicated that basic self-disturbance scores were higher in schizophrenia spectrum cases, irrespective of transition to psychosis, than nonschizophrenia spectrum cases.</AbstractText>
<AbstractText Label="DISCUSSION" NlmCategory="CONCLUSIONS">The results indicate that identifying basic self-disturbance in the UHR population may provide a means of further "closing in" on individuals truly at high risk of psychotic disorder, particularly of schizophrenia spectrum disorders. This may be of practical value by reducing inclusion of "false positive" cases in UHR samples and of theoretical value by shedding light on core phenotypic features of schizophrenia spectrum pathology.</AbstractText>
</Abstract>
<AuthorList CompleteYN="Y">
<Author ValidYN="Y">
<LastName>Nelson</LastName>
<ForeName>Barnaby</ForeName>
<Initials>B</Initials>
<AffiliationInfo>
<Affiliation>Orygen Youth Health Research Centre, University of Melbourne, 35 Poplar Road (Locked Bag 10), Parkville, Victoria 3052, Australia. nelsonb@unimelb.edu.au</Affiliation>
</AffiliationInfo>
</Author>
<Author ValidYN="Y">
<LastName>Thompson</LastName>
<ForeName>Andrew</ForeName>
<Initials>A</Initials>
</Author>
<Author ValidYN="Y">
<LastName>Yung</LastName>
<ForeName>Alison R</ForeName>
<Initials>AR</Initials>
</Author>
</AuthorList>
<Language>eng</Language>
<PublicationTypeList>
<PublicationType UI="D016428">Journal Article</PublicationType>
<PublicationType UI="D013485">Research Support, Non-U.S. Gov't</PublicationType>
</PublicationTypeList>
<ArticleDate DateType="Electronic">
<Year>2012</Year>
<Month>02</Month>
<Day>20</Day>
</ArticleDate>
</Article>
<MedlineJournalInfo>
<Country>United States</Country>
<MedlineTA>Schizophr Bull</MedlineTA>
<NlmUniqueID>0236760</NlmUniqueID>
<ISSNLinking>0586-7614</ISSNLinking>
</MedlineJournalInfo>
<CitationSubset>IM</CitationSubset>
<MeshHeadingList>
<MeshHeading>
<DescriptorName UI="D000293" MajorTopicYN="N">Adolescent</DescriptorName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D016022" MajorTopicYN="N">Case-Control Studies</DescriptorName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D018450" MajorTopicYN="N">Disease Progression</DescriptorName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D005260" MajorTopicYN="N">Female</DescriptorName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D008137" MajorTopicYN="N">Longitudinal Studies</DescriptorName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D008297" MajorTopicYN="N">Male</DescriptorName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D062706" MajorTopicYN="Y">Prodromal Symptoms</DescriptorName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D011379" MajorTopicYN="N">Prognosis</DescriptorName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D016016" MajorTopicYN="N">Proportional Hazards Models</DescriptorName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D011618" MajorTopicYN="Y">Psychotic Disorders</DescriptorName>
<QualifierName UI="Q000175" MajorTopicYN="N">diagnosis</QualifierName>
<QualifierName UI="Q000523" MajorTopicYN="N">psychology</QualifierName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D012044" MajorTopicYN="N">Regression Analysis</DescriptorName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D012559" MajorTopicYN="N">Schizophrenia</DescriptorName>
<QualifierName UI="Q000175" MajorTopicYN="Y">diagnosis</QualifierName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D012565" MajorTopicYN="Y">Schizophrenic Psychology</DescriptorName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D012649" MajorTopicYN="Y">Self Concept</DescriptorName>
</MeshHeading>
<MeshHeading>
<DescriptorName UI="D055815" MajorTopicYN="N">Young Adult</DescriptorName>
</MeshHeading>
</MeshHeadingList>
</MedlineCitation>
<PubmedData>
<History>
<PubMedPubDate PubStatus="entrez">
<Year>2012</Year>
<Month>2</Month>
<Day>22</Day>
<Hour>6</Hour>
<Minute>0</Minute>
</PubMedPubDate>
<PubMedPubDate PubStatus="pubmed">
<Year>2012</Year>
<Month>2</Month>
<Day>22</Day>
<Hour>6</Hour>
<Minute>0</Minute>
</PubMedPubDate>
<PubMedPubDate PubStatus="medline">
<Year>2013</Year>
<Month>5</Month>
<Day>17</Day>
<Hour>6</Hour>
<Minute>0</Minute>
</PubMedPubDate>
</History>
<PublicationStatus>ppublish</PublicationStatus>
<ArticleIdList>
<ArticleId IdType="pubmed">22349924</ArticleId>
<ArticleId IdType="pii">sbs007</ArticleId>
<ArticleId IdType="doi">10.1093/schbul/sbs007</ArticleId>
<ArticleId IdType="pmc">PMC3494062</ArticleId>
</ArticleIdList>
<ReferenceList>
<Reference>
<Citation>Acta Psychiatr Scand. 2003 Aug;108(2):126-33</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">12823169</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Psychopathology. 2009;42(6):361-9</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">19752589</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Acta Psychiatr Scand. 2006 Apr;113(4):247-72</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">16638070</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Arch Gen Psychiatry. 2001 Feb;58(2):158-64</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">11177117</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Schizophr Bull. 2011 Mar;37(2):344-51</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">19528205</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Schizophr Bull. 2008 Mar;34(2):381-92</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">17702990</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Br J Psychiatry Suppl. 2005 Aug;48:s49-54</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">16055808</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Am J Psychiatry. 1992 Sep;149(9):1148-56</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">1386964</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Schizophr Res. 2003 Mar 1;60(1):21-32</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">12505135</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Aust N Z J Psychiatry. 2009 Feb;43(2):118-28</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">19153919</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Schizophr Bull. 2011 Sep;37(5):1017-26</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">20176859</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Psychopathology. 2009;42(5):283-92</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">19609098</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Aust N Z J Psychiatry. 2005 Nov-Dec;39(11-12):964-71</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">16343296</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Arch Gen Psychiatry. 2008 Jan;65(1):28-37</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">18180426</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Schizophr Res. 2006 Jan 1;81(1):83-90</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">16297599</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Neurosci Biobehav Rev. 2009 Jun;33(6):807-17</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">19428493</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Psychopathology. 2011;44(6):386-90</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">21847006</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Schizophr Bull. 2000;26(1):217-32</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">10755683</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Early Interv Psychiatry. 2009 May;3(2):83-93</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">21352181</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>World Psychiatry. 2011 Oct;10(3):200-4</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">21991279</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Aust N Z J Psychiatry. 1996 Oct;30(5):587-99</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">8902166</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Arch Gen Psychiatry. 1984 Nov;41(11):1050-6</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">6497567</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Psychopathology. 2005 Sep-Oct;38(5):259-67</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">16179812</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Schizophr Bull. 2007 May;33(3):673-81</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">17404389</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Compr Psychiatry. 2003 Mar-Apr;44(2):121-34</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">12658621</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Schizophr Bull. 2011 Nov;37(6):1121-30</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">21771902</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Psychopathology. 2005 Sep-Oct;38(5):236-58</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">16179811</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Schizophr Bull. 2011 Nov;37(6):1229-47</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">20418446</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Schizophr Bull. 2009 Nov;35(6):1034-6</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">19478239</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Med Hypotheses. 2010 Jan;74(1):186-8</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">19665853</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Schizophr Bull. 2007 Jan;33(1):108-12</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">17158191</ArticleId>
</ArticleIdList>
</Reference>
<Reference>
<Citation>Schizophr Bull. 2003;29(3):427-44</Citation>
<ArticleIdList>
<ArticleId IdType="pubmed">14609238</ArticleId>
</ArticleIdList>
</Reference>
</ReferenceList>
</PubmedData>
</pubmed>
<affiliations>
<list>
<country>
<li>Australie</li>
</country>
<region>
<li>Victoria (État)</li>
</region>
<settlement>
<li>Melbourne</li>
</settlement>
<orgName>
<li>Université de Melbourne</li>
</orgName>
</list>
<tree>
<noCountry>
<name sortKey="Thompson, Andrew" sort="Thompson, Andrew" uniqKey="Thompson A" first="Andrew" last="Thompson">Andrew Thompson</name>
<name sortKey="Yung, Alison R" sort="Yung, Alison R" uniqKey="Yung A" first="Alison R" last="Yung">Alison R. Yung</name>
</noCountry>
<country name="Australie">
<region name="Victoria (État)">
<name sortKey="Nelson, Barnaby" sort="Nelson, Barnaby" uniqKey="Nelson B" first="Barnaby" last="Nelson">Barnaby Nelson</name>
</region>
</country>
</tree>
</affiliations>
</record>

Pour manipuler ce document sous Unix (Dilib)

EXPLOR_STEP=$WICRI_ROOT/Bois/explor/PoplarV1/Data/Main/Exploration
HfdSelect -h $EXPLOR_STEP/biblio.hfd -nk 002B89 | SxmlIndent | more

Ou

HfdSelect -h $EXPLOR_AREA/Data/Main/Exploration/biblio.hfd -nk 002B89 | SxmlIndent | more

Pour mettre un lien sur cette page dans le réseau Wicri

{{Explor lien
   |wiki=    Bois
   |area=    PoplarV1
   |flux=    Main
   |étape=   Exploration
   |type=    RBID
   |clé=     pubmed:22349924
   |texte=   Basic self-disturbance predicts psychosis onset in the ultra high risk for psychosis "prodromal" population.
}}

Pour générer des pages wiki

HfdIndexSelect -h $EXPLOR_AREA/Data/Main/Exploration/RBID.i   -Sk "pubmed:22349924" \
       | HfdSelect -Kh $EXPLOR_AREA/Data/Main/Exploration/biblio.hfd   \
       | NlmPubMed2Wicri -a PoplarV1 

Wicri

This area was generated with Dilib version V0.6.37.
Data generation: Wed Nov 18 12:07:19 2020. Site generation: Wed Nov 18 12:16:31 2020